Population pharmacokinetic modelling of Emfilermin in healthy postmenopausal women and in women undergoing Embryo Tranfer (ET)

Nguyen T. X. Q.(1), Munafo A. (1), Goggin T. (1)

(1) Serono International S.A, Geneva, Switzerland

Background: Current evidence suggests that endogenous leukaemia inhibitory factor (LIF) – a heavily glycosylated cytokine plays a physiological role in embryo implantation (1,2). Clinical trials with recombinant human leukaemia inhibitory factor (r-hLIF – obtained in E. coli and consequently non glycosylated) are in progress to assess its potential therapeutic role in overcoming embryo implantation failure following IVF (In Vitro Fertilisation) and ET.

Purpose: The purpose of this analysis was to describe the pharmacokinetics of recombinant-hLIF (Emfilermin; AMRAD Corp. Ltd, Australia) in healthy postmenopausal women and in women with recurrent implantation failure (RIF) who undergo IVF and ET.

Methods: Data from the following studies were combined.
Study 1 :  was a double-blind, randomised, placebo-controlled, phase I study in twelve healthy oestrogenised postmenopausal women (mean age: 57 ± 5 years). Subjects received 150 µg of Emfilermin or placebo SC twice daily during 7 days. Frequent pharmacokinetic sampling was performed on day 1 and 7.
Study 2 : was a double-blind, randomised, placebo-controlled, proof of concept study in sixty-six young women with RIF justifying IVF and ET (mean age: 33 ± 2 years). Patients received 150 µg of Emfilermin or placebo SC just before ET and twice daily for 7 days. Pharmacokinetic sampling was sparse and was done on Days 1, 4 and 7.
Non-compartmental analysis on serum LIF concentrations was performed using WinNonLin version 4.0. Population pharmacokinetic analysis was performed using NONMEM version V. 

Results: Non-compartmental analysis of data of Study 1 showed an apparent terminal half-life at 2.5 h (median value), which was similar on day 1 and day 7. An accumulation ratio of 1.3-1.5 was observed in 4 out of 10 subjects, unexplained by the observed half-life. The reason for this could not be determined.

The subsequent population pharmacokinetic analysis was performed on (assumed) steady-state data only (day 4 and day 7). A one-compartment
disposition model with zero order input was used. The duration of the absorption was 1.1 h. Inter-subject variability on this parameter was negligible. The apparent volume of distribution was 184 L (with a CV of 28%)and devoid of any significant covariate effects. The observed CL/F on Study Day 7 was 31% higher compared to the Study Day 4 (Study Day modelled as a covariate). In addition the systemic clearance in younger women with RIF was reduced by 40% compared to that in older healthy women (which was 61.3 L/h with a CV of 11%). CL/F was also found to be roughly proportional to body weight, with a 2?fold increase in body weight resulting in a 1.9-fold increase in CL/F.

The geometric mean post-hoc estimates of apparent clearance and volume of distribution and their variability were consistent with the population estimates. In healthy subjects, results were fully consistent with those obtained using non-compartmental methods. The residual variability on r-hLIF serum levels was quite low (CV = 17%).


Conclusion: Recombinant –hLIF pharmacokinetics are quite complex. The initial assumption of steady-state is not supported by the results of this analysis, which suggests possible time dependence in the apparent clearance of r-hLIF,higher on Day 7 compared to Day 4. All available pharmacokinetic data will now be combined and analyzed to further characterize this possible phenomenon. The clinical impact of this non-stationarity is however mitigated by the fact that the observation period in this experiment corresponds to the intended duration of treatment (7 days)
in this indication.


References:
1- Laird, S.M., Tuckerman, E.M., Dalton, C.F., Dunphy, B.C., Li, T.C. and Zhang, X. (1997)  The production of human leukemia inhibitory factor by human endometrium: presence in uterine flushings and production by cells in culture.  Human Reproduction 12, 569-574.
2- Hambartsoumian, E. (1998)  Endometrial leukemia inhibitory factor (LIF) as a possible cause of unexplained infertility and multiple failures of implantation.  American Journal of Reproductive Immunology 39, 137-143.

Reference: PAGE 12 (2003) Abstr 384 [www.page-meeting.org/?abstract=384]

Poster: poster